neuroleptic malignant syndrome vs malignant hyperthermia

The RIGID patient: tetanus mimics - Serotonin Syndrome ... | GrepMedCOVID-19 is an emerging and rapidly evolving situation. Account name Save an appointment to archive E-mail citation Add to Collections Add to My Bibliography Your search saved Create a file for external dating management software Your RSS Feed Full text links Actions Share Page navigation Neuroleptic malignant syndrome and malignant hyperthermia: end of a controversy? Membership Neuroleptic malignant syndrome and malignant hyperthermia: end of a controversy? Authors Membership Summary Two primary hypotheses have been proposed to explain the physiopathology of malignant neuroleptic syndrome (NMS): 1) that NMS is produced by abrupt and extensive blockage of central dopamine receptors by neuroleptics, particularly in nigrostriatal and hypotallic pathways; and 2) that NMS, such as malignant hyperthermia (MH), results from a presenexistent defect in skeletal muscle metabolism. To evaluate these models, the authors review the studies published since 1980 of clinical characteristics, epidemiology, risk factors, laboratory evaluation and relevant animal models of NMS and MH. Data from these studies suggest that although NMS and MH are clinically similar, they are pharmacologically different, which means that a cross-reaction between triggering agents is unlikely to occur. Similar articles Cited by 6 articles Types of publication MeSH Terms Substances Related information Link Out - more resources Total Text SourcesMedicalNCBI Literacy resources National Library of Medicine

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little white coats: Serotonin syndrome, neuroleptic malignant syndrome, anticholinergic toxidrome and malignant hyperthermia can present similarly with hyperthermia, tachycardia, confusion and hypertension. What clinical characteristics help ...

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